- cross-posted to:
- upliftingnews@lemmy.world
- cross-posted to:
- upliftingnews@lemmy.world
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I remember when I was about 8 I asked my dad (who had the typical cul de sac bald on top thing going, and was a general practitioner) why he didn’t use any of the hair growth pills. He told me that he would “rather have a dick that worked and no hair, than a full head of hair and a limp dick”.
I still kinda wish he just told me “there are side effects”, I was fucking 8.
They’re called testosterone blockers.
I look forward to hearing how this will fuck you up 20 years down the road. Just bite the bullet, grow a pair, and let that hair go. You’ll thank yourself down the road. Note, that I’m referring to men specifically.
It’s harder for us girls. I’ve got a hat with a wig sewn in so I have that going for me which is nice.
Minodixil has already shown dangerous side effects, or so I read somewhere (but IIRC it was a reputable source, I just don’t have a link to prove it, sorry).
No need to wait that long. Hair loss drugs fuck with your androgens and can cause depression
fuck with your androgens and cause depression
chases daily fin pill with the double daily 2.5mg of sublingual estradoil
Andro-who-now?
Seriously though, some of us are glad to see em go lol.
Fair but that is a small minority of people
Oh yeah no its just that between me being better off without em, and the horrific gender dysphoria I felt from a receding hairline make that shit laughable.
I def think buzzing is the right answer for a lotta dudes.
What if I’m already bald and struggle with depression?
Stick to the shampoo version. Finasteride is the really notorious one though
No, I like my hair.
There are some people that look good bald. I am not one of them.
We’ve got wars and bad economy everywhere in what is quickly becoming a full on dystopia but we have a pill that can give you your boner back and now another pill that will give you your hair back. You can then pretend you are an “alpha” again and do more wars and cheat the masses out of more money. Pharmacy is a miracle! /s
What a silly thing to say. No need to shame men for taking these pills. I for one love that there are hair regrowth pills since its gonna help a lot with my mtf transition. Should they stop development into these pills just because there are some dicks in the whitehouse?
Forgot about the pill that will make you stop eating.
I’m on oral minox, finn, and estrogen. My hair regrowth is ridiculous.
Would you be open to talking about this with me? I’m about to start estrogen and very curious about this combo.
My hairline came back on fin+estradoil.
Started on 1mg oral fin and 2mg of daily oral estradoil so fin was doin good in its own.
Now I’m on 5mg sublingual e, and fuck my hairs probably back to like 22 year old levels.
Still miss the 19 -year-old-maine thick though lol.
Keep in mind I started fucking w/ hairloss stuff in early 20s before my egg cracked.
You gotta catch it when it starts for really good results from what I know.
Ty, will DM later when I’m home.
I’m happy to, but I’m not super far into my journey. I just have a huge increase in miniaturised hairs regrowing in my crown and M shape. I’m still waiting for them to go terminal and thicken.
Is there anything in particular you wanted to know? Also be warned oral minox will cause body hair increase.
Is it okay if we go somewhere more trans to chat about this? I can make a post on blahaj and tag you.
For sure! LW isn’t the most accommodating space. Feel free to message me as well if you want to keep it private.
I’m gonna be so fucking hairy.
Seriously, though, pro-tip, I got my last 3-month batch of the regular Minoxidil at Ollie’s in the US for $8. YMMV. But $8 is a steal.
There have been reports of water forming around the heart due to oral minoxidil. It needs to be tested that the oral extended release version is safe. The problem is only a small percentage might have this potentially lethal side effect, so it needs to be tested very throughly
Minoxidil is also a super potent blood pressure medicine. I don’t have the numbers obviously, but this is already widely available at compounding pharmacies so I imagine there are probably thousands of people already using it.
It has a black box warning about potentially fatal side effects. Everyone using it is using it off label for regrowing hair and trying to micro dose it. There are plenty of safer blood pressure medications. The hope is that by using extended release the blood concentration never peaks.
that is basically a low dose hypertension drug lol and also coincidentally exactly how minoxicidil spray was discovered when they realized some hypertension drugs resulted in hair growth. seems like it reached a full circle.
My limited research into these is that you pretty much have to keep taking them. I considerd it, mostly because of sun and skin cancer risks, but not if I have to take a drug for the rest of my life. I have hats, they work fine.
Lot of misinformation in this thread. Every drug has side effects but that doesn’t mean everyone or even a majority of people will experience them.
Minoxidil was originally designed as a blood pressure med and was found to support hair growth. Kind of like Viagra was designed for pulmonary hypertension and it was later found that it supported erections.
The doses of minoxidil given to support the anagen or thicker phase of hair growth {2.5 to 5 mg) are much lower than those given for blood pressure (10 to 20 mg). The higher doses are more commonly associated with side effects like lightheadedness, palpitations, lower extremity edema and pericardial effusion.
Detasteride or Finasteride decrease the conversion of testosterone to dihydrotestosterone. Scalp DHT accelerates follicle minituarization. This medication has a long half life so if youve taken it for weeks it stays in your system for weeks even after youve stopped it. There are people that have reported longer standing mood changes due to this drug.
I know many who tolerate both just fine but everyone should assess their own risk tolerance and make a decision based on that. The topical formulations help but the oral combination is much more effective and for those that pursue a hair transplant these meds will always be started afterwards (often beforehand as well) because otherwise your DHT will just wipe out the transplanted follicles.
Yep, take a pill that goes in your stomach, absorbed through the intestines, into your blood stream, liver, etc, to grow hair on your head.
Bullshit.
Oh look, an amateur pharmacologist.
This is why we grind aspirins into our forehead to relieve a headache.
Tell us more about how you don’t understand how medication works?
There is good reason to limit off-target drug delivery, to minimize side effects in tissues that don’t need exposure to the drug.
- Is the oral route more effective than topical?
- What are the systemic effects of the oral med?
- Was the study quoted in the article in any way funded by a pharmaceutical company that would be granted an exclusive license for a “new formulation,” or is it trustworthy?
Do you? We have no idea how much of the medication works. We know it works in some parts better than others through observations and assumptions.
If we actually knew medicine, we tackle pain, injuries and other ailments through localised medical intervention, not taking a pill and hoping for the best.
Dude, you’re literally describing pharmacology.
Localized effects have always been the goal, but actually making a drug that gets only to its intended destination, does a very specific thing, and doesn’t escape to the rest of the body is very hard, very expensive, very painful, or a combination of the three.
We actually do not know how medication works.
You’d be baffled knowing how we research new medications (it’s randomly trying millions of different molecules).
One breakthrough where we know more is MRNA but that’s just a small part.
And metabolism? We have a poor understanding of about 10-15%(we don’t even know where it all happens or how much there is), the rest is unknown.
Oh shut up. Every drug has a defined target.
Yes but we still start out with millions of molecules for each target. So shut up I guess?
Is everyone in this sub thread 11 years old?
I meaaan, Paracetamol goes through the same process and it lowers headaches.
Another example:
Ozempic gets injected into subcutaneous fat and works its way through the body, via the liver and heart, to alter the glp hormones in your pancreas and brain.
I’m sure we could think of lots of other examples where drugs are administered somewhere else to affect different areas.
OPs argument is as ignorant as flat-earth theorists.
Edit: and this shows this thread is a sausage-fest, as nobody mentioned either the pill or the morning-after pill which I’m sure most women would have probably said instantly.
Whoever takes Ozempic is basically a lab rat at this stage. No short term issues have been observed but we don’t know the long term effects.
All medication has the possibility of side effects.
In the 20 years GLP-1 medication has been used by type-2 diabetes patients (because that’s who they were really for), there have been more positives than negatives. Simple as that. There are side effects but those are better than the well-documented side effects of type-2 (e.g. cardiovascular disease, kidneys, eyes and feet, including the possibility of amputation. And that’s before we get into the social stigma of being morbidly obese, the mental health issues associated with that and the social isolation that often comes with it).bb
The. You just proved my point, we have no idea how paracetamol actually works, after 130years of study.
https://www.livescience.com/health/we-may-finally-know-how-tylenol-works-and-its-not-how-we-thought
If we don’t know how those processes work but we know the effect they cause, how can you so confidently say that the hair growth thing is BS? Knowing how it works is besides the point, if we know the effect it causes and it being ingested, how can you call bullshit to another thing having an effect in a different part of our body than where it’s introduced?
I didn’t prove your point and I’m sorry to learn you think I did.
And you shouldn’t munch them too often because they do go through the liver etc.
OP has a point IMO.
What does that have to do with the fact that a pill being ingested has no direct relation with the place it has an effect on? That’s the point I was trying to imply, thus countering the point OP was making.
IMO OP has a point as the delivery mechanism already exists (smearing it on the head) so making it take the longer way through our inner organs is, if not “bullshit” probably less good, as just smearing it has proven dangerous.
Maybe I just think nobody can be that stupid and believe eating a pill can’t do something to the head.
Yes, take a pill that goes to your stomach, absorbed by your intestines, into your bloodstream, liver, etc. to decrease your acne (isotretinoin)
… to stop asthma (salbutamol)
etc.
APLLY DIRECTLY TO YOUR FOREHEAD! APPLY DIRECTLY TO YOUR FOREHEAD!
You say that but oral/sublingual fin and estradoil worked much better than topical Finn/minoxidil alone.
That includes when I was still taking estradoil orally.
It was originally for high blood pressure if I remember correctly. Side effect was unwanted hair growth. Very quickly became a topical for wanted hair growth.
I heard that minoxidil doesn’t actually grow hair.
Minoxidil: Increases blood flow by dilating blood vessels. More blood flow means more resources for hair in the region that the minoxidil is acting in. More resources = hair grows thicker. CANNOT grow hair for follicles that are dead. Effects go away when you stop taking it.
Finestaride (probably the one you’re talking about): blocks DHT that causes male pattern baldness. Prevents hair loss caused by DHT (for most people). Effects last only while you take it.
Hence, people generally combine these things. Finestaride to stop mpb/lessen its pace, while minoxidil to make fine hairs at the hairline grow thicker.
Minoxidil extends the Anagen (growth) phase of the hair cycle and increases the size of miniaturized follicles. While it doesn’t create brand-new follicles, it transforms thin, resting hairs into thicker, more pigmented ones, improving the density and appearance of the hair you already have.
Correct, it’s blocking dht which enables your hair follicles to grow hair. If the Minoxidil grew the hair it would just fall out again ✨
Incorrect, that is finesteride.
God damn me.
Thanks. It work good, still.
